Traditional medical approaches to fostering bone health is to reduce the activity of the osteoclasts. Osteoclasts are the cells that resorb, or break down and absorb, bone tissue back into the body. Osteoclasts are highly specialized cells that must work in perfect synchronization with osteoblasts to maintain the skeletal system. Unfortunately allopathic drugs in reducing the osteoclast activity has the unfortunate effect of not ony preventing bone loss, but also does not foster the renewal of bone. Therefore, the body will be subjected to the increased possibility of microfactures and other factors that weaken bones.
We have found that calcium (particularly eggschell calcium) can improve bone marrow density, but it must be used in conjunction with other co-factors like collagen protein (silicon supplements support this), vitamin K-2 menaquinone-7 and magnesium. The K2 will activate osteocalcin (bone gla protein) a non-collagen protein abundant in bone. Magnesium has been shown to keep calcium in the bones and must be balanced with calcium intake in a 1:1 ratio. In addition a good electrolyte-forming trace-mineral supplement, (www.electroblast.com) that includes boron, manganese, copper, and silica is needed to help remineralize the bone. Vitamin D is well-known as a hormone involved in mineral metabolism and bone growth. It facilitates intestinal absorption of calcium, although it also stimulates absorption of phosphate and magnesium ions. In the absence of vitamin D, dietary calcium is not absorbed at all efficiently. Therefore, vitamin D3 (Cholecalciferol) supplmentation should be added to your diet if you do not get enough sun nor eat fish. D3 is the natural form of D. D2 is synthetic so we advise going natural.
Information extracted from: K.M. Rynder; “Magnesium, etc.” Jnl of the Am. Geriatrics Soc. 53(11), 1875-80 (2005); K.J. Ruff, “Eggshell, etc.” Clin. Interv. in Aging 4, 235-240 (2009)
Your observations about: “Traditional medical approaches to fostering bone health is to reduce the activity of the osteoclasts…” is absolutely correct. A better, natural means towards bone health is to use natural, bio-identical progesterone.
Visit this website to learn more: http://www.ProgestaPlus.com.
In their FAQ sections read:
How do estrogen and progesterone effect bone health?
Bones, like skin, blood and hair, are living growing organs. Though much slower growing, bones do have a cycle of repair, replacement and growth. There are two functioning cell groups that take care of bone maintenance and health; ‘Osteoclasts’ and the ‘Osteoblasts’. Osteoclasts are little ‘PacMen’ like cells that remove old, worn-out, crystallized bone cells. Behind them come the osteoblasts, which repair and rejuvenate bones using Vitamin D and over 17 different minerals (not just calcium!). Osteoporosis is not considered a ‘disease’ as much as a nutritional and hormonal deficiency condition. According to the research of Dr. John Lee* and Dr. Jerilynn C. Prior (re: Sherrill Sellman’s Hormone Heresy book) Estrogen does not benefit osteoporosis conditions. Estrogen retards the rate of measurable bone loss by inhibiting the osteoclasts removal of old bone tissue but does not reverse or cure osteoporosis (leading to higher rates of fractures in women who seem to be reducing osteoporosis with synthetic hormones). Though some studies have shown that a lack of estrogen increases a substance called interleukin-6 (associated with bone growth and loss), Dr. Lee’s research and patient follow-up studies demonstrate that the effectiveness of estrogen in reducing bone loss is only noticeable during the five years following menopause. After that five-year period, estrogen is not effective in moderating bone loss (which continues at the same rate as in those women not using any estrogen supplementation). All the unbiased evidence supports the obvious fact that the actual reduced rate of bone loss maybe relevant to the levels of progesterone, not estrogen. Dr. Lee’s* research, and that of Dr. Jerilynn Prior, confirm that the missing factor in bone loss is low levels of progesterone. Supplementing with natural Progesterone stimulates osteoblast activity in building new bone tissue and mass in bone areas where healthy active osteoclasts had removed worn out bone cells. The book, Hormone Heresy, reports the results of a three-year study of 63 post-menopausal women with osteoporosis. These women, using transdermal progesterone cream, experienced an average 7 to 8 per cent bone-mass density increase in the first year, 4 to 5 per cent in the second year, and 3 to 4 per cent in the third year. Untreated women in this age category typically lose 1.5 per cent bone-mass density per year. Such results have not been found with any other form of hormone replacement therapy or dietary supplementation. A remarkable finding of Dr. Lee’s* research is that natural progesterone not only prevents osteoporosis, it will actually reverse it! Dr. Lee* (and subsequent research findings) have confirmed that women using a 20 mg progesterone skin cream dose a day, saw a 15% increase of bone mineral density in the first year with and total of 30% bone mass increase over 3 years. Osteoblast cells (responsible for making new bone) do have progesterone receptors but do not have estrogen receptors. This means that progesterone (the natural form, not the synthetic progestins) is responsible, for building bone tissue, not estrogen.
To further support and enhance progesterone’s ability to treat osteoporosis, Dr. Lee* recommends a low-protein diet with leafy greens, legumes and whole grains. One should avoid sodas and limit alcohol consumption. Supplement with Vitamin C, B-6, D, Beta-carotene, magnesium, Zinc, Calcium and routine exercise, 30 minutes/day three times a week
Bone health is of course at best if you take lots of calcium and magnesium supplements. Your diet can be a great source of calcium too. `.:,`
Warm regards http://www.foodsupplementdigest.com“>
About 18 million Americans have osteopenia. Osteopenia refers to early signs of bone loss that can turn into osteoporosis. With osteopenia, bone mineral density is lower than normal. However, it is not yet low enough to be considered osteoporosis.:
Please do go look at this useful web-site